Gladstone in the News
The Gladstone Institutes is gratified to receive media attention from around the globe. Check out the highlights of recent press coverage of Gladstone scientists and research. For other news, please be sure to follow us on Facebook and Twitter.
A pair of landmark discoveries made 40 years apart have earned the 2012in Physiology or Medicine for John B. Gurdon of the University of Cambridge in England and Shinya Yamanaka of the Gladstone Institutes and Kyoto University in .
Shinya Yamanaka, PhD, MD, it was announced today, has just won a much-deserved Nobel Prize in Medicine for an astonishing discovery.
John B. Gurdon transferred DNA between a tadpole and a frog to clone the first animal. Shinya Yamanaka used Gurdon’s concept to turn ordinary skin into potent stem cells. Both won the Nobel Prize for medicine today.
Shinya Yamanaka could have made bits of sewing machines for a living. Instead, his tinkering with the building blocks of life has made him a Nobel Prize winner.
The Nobel Committee awarded this year’s prize in Physiology or Medicine to John Gurdon, of the University of Cambridge, UK and Shinya Yamanka, of the Gladstone Institutes and the University of Kyoto, Japan for “for the discovery that mature cells can be reprogrammed to become pluripotent,” this morning in Stockholm.
Dr. Shinya Yamanaka of Japan is with the Gladstone Institutes shares the Nobel Prize in medicine with Britain's John Gurdon.
Gladstone researchers say the discovery could have a profound impact on how we treat hearts and heart disease.
Retired Senior Investigator Karl Weisgraber has had a long, difficult recovery from a fall off a ladder that caused a traumatic brain injury last October.
Scientists at the Gladstone Institutes have created the first "genomic blueprint" of the human heart, unveiling the exact order and timing of genetic events that must take place for an embryonic heart cell to become a beating, life-sustaining organ.
Gladstone investigators Eric Verdin, MD, and Shomyseh Sanjabi, PhD, are researching why patients living with HIV are aging prematurely.